Search results for "Enterochromaffin Cells"

showing 10 items of 10 documents

Short- and long-term effects of vinblastine on the rat adrenal medulla

1979

The effects of a single high dose (10mg/kg) of vinblastine (vb) sulfate (“Velbe”, Lilly) on the ultrastructure, catecholamine (CA) content and activity of CA-synthesizing enzymes of the rat adrenal medulla were studied for up to 120h after intravenous injection of the drug.

medicine.medical_specialtyTime FactorsHistologyChemical PhenomenaVinblastinePathology and Forensic MedicineInternal medicineSingle high doseEnterochromaffin CellsmedicineAnimalsCells Culturedchemistry.chemical_classificationCell BiologyMolecular medicineRatsVinblastineChemistryEnzymemedicine.anatomical_structureEndocrinologychemistryAdrenal MedullaUltrastructureCatecholamineFemaleAdrenal medullamedicine.drugCell and Tissue Research
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Cisplatin increases the release of 5-hydroxytryptamine (5-HT) from the isolated vascularly perfused small intestine of the guinea-pig: Involvement of…

1991

Isolated segments of the guinea-pig small intestine were vascularly perfused and the release of 5-hydroxytryptamine (5-HT) and 5-hydroxyindoleacetic acid (5-HIAA) into the portal venous effluent determined by high pressure liquid chromatography with electrochemical detection. Release of acetylcholine from isolated superfused intestinal segments was determined as outflow of [3H]radioactivity from preparations preincubated with [3H]choline. Cisplatin (3 microM) increased the outflow of 5-HT and 5-HIAA by about 90%. At 30 and 100 microM cisplatin decreased the outflow of 5-HT and its metabolite by 40%-50%. The stimulatory effect of cisplatin was consistently observed only when the bicarbonate-…

MaleSerotoninmedicine.medical_specialtymedicine.drug_classMetaboliteGuinea PigsTetrodotoxinIn Vitro Techniqueschemistry.chemical_compoundInternal medicineIntestine SmallEnterochromaffin CellsmedicineAnimalsReceptor5-HT receptorPharmacologyCisplatinDose-Response Relationship DrugImidazolesGeneral MedicineHydroxyindoleacetic AcidReceptor antagonistOndansetronAcetylcholineSmall intestinePerfusionEndocrinologymedicine.anatomical_structurechemistryReceptors SerotoninFemaleHexamethoniumCisplatinAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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NK1- and NK3-receptor mediated inhibition of 5-hydroxytryptamine release from the vascularly perfused small intestine of the guinea-pig

1997

The effects of tachykinins on the spontaneous release of 5-hydroxytryptamine (5-HT) from the enterochromaffin cells into the portal circulation was investigated in vitro using the vascularly perfused isolated guinea-pig small intestine. 5-HT was determined by HPLC with electrochemical detection. Test substances were applied intraarterially. Substance P (SP) caused a concentration-dependent decrease in 5-HT outflow with an EC50 of 50 pmol/l. Similarly, the selective NK1 receptor agonist SP methyl ester (1 nmol/l) significantly inhibited 5-HT outflow (to 51 +/- 3%). When tetrodotoxin (1 mumol/l) was added to the arterial perfusion medium, the inhibition by SP of 5-HT outflow was not affected.…

MaleAgonistSerotoninmedicine.medical_specialtymedicine.drug_classGuinea PigsStimulationSubstance PTetrodotoxinSubstance Pchemistry.chemical_compoundNeurokinin-1 Receptor AntagonistsIleumInternal medicineEnterochromaffin CellsmedicineAnimalsDrug InteractionsReceptorPharmacologyChemistryReceptors Neurokinin-3General MedicineMolecular biologyPeptide FragmentsSmall intestineEndocrinologymedicine.anatomical_structureTetrodotoxinEnterochromaffin cellNK1 receptor antagonistNaunyn-Schmiedeberg's Archives of Pharmacology
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Effects of cigarette smoking or ingestion of nicotine on platelet 5-hydroxytryptamine (5-HT) levels in smokers and non-smokers.

1992

Platelets of healthy smokers and non-smokers were prepared and their content of 5-hydroxytryptamine was determined by HPLC with electrochemical detection. Platelet 5-HT levels in smokers (728 +/- 156 pmol per 10(8) platelets, mean +/- SEM, n = 9) were significantly higher than those in non-smokers (353 +/- 156 pmol per 10(8) platelets, n = 11). Smoking of a single cigarette caused a transient increase in platelet 5-HT levels by about 350% in non-smokers, but had no additional effect in smokers. Similarly, chewing of nicotine gum (4-8 mg nicotine) resulted in a transient increase in platelet 5-HT by about 100% in non-smokers, but not in smokers. In conclusion, smoking of cigarettes can cause…

AdultBlood Plateletsmedicine.medical_specialtyNicotineSerotoninAdministration OralReceptors NicotinicNicotine03 medical and health sciences0302 clinical medicineCigarette smokingInternal medicineDrug DiscoverymedicineEnterochromaffin CellsIngestionHumansPlateletReceptorGenetics (clinical)5-HT receptorbusiness.industrySmokingGeneral Medicinerespiratory tract diseases3. Good healthEndocrinologyNicotine gum030220 oncology & carcinogenesisReceptors Serotoninbehavior and behavior mechanismsMolecular MedicineSerotoninbusiness030217 neurology & neurosurgerymedicine.drugThe Clinical investigator
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Control of Gastric Acid Secretion in Somatostatin Receptor 2 Deficient Mice: Shift from Endocrine/Paracrine to Neurocrine Pathways

2007

The gastrin-enterochromaffin-like (ECL) cell-parietal cell axis is known to play an important role in the regulation of gastric acid secretion. Somatostatin, acting on somatostatin receptor type 2 (SSTR2), interferes with this axis by suppressing the activity of the gastrin cells, ECL cells, and parietal cells. Surprisingly, however, freely fed SSTR2 knockout mice seem to display normal circulating gastrin concentration and unchanged acid output. In the present study, we compared the control of acid secretion in these mutant mice with that in wild-type mice. In SSTR2 knockout mice, the number of gastrin cells was unchanged; whereas the numbers of somatostatin cells were reduced in the antru…

medicine.medical_specialtyendocrine systemCell CountGalaninBiologyHistidine DecarboxylaseArticleGastric AcidMiceEndocrinologyParietal Cells GastricInternal medicineParacrine CommunicationmedicineEnterochromaffin CellsSomatostatin receptor 2AnimalsReceptors SomatostatinEnterochromaffin-like cellGastrinMice KnockoutDelta cellSomatostatin receptorGastrin-Secreting Cellsdigestive oral and skin physiologyNeurosecretory SystemsMicroscopy ElectronEndocrinologySomatostatinGastric MucosaVesicular Monoamine Transport ProteinsG cellReceptors Galaninhormones hormone substitutes and hormone antagonists
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Rotavirus stimulates release of serotonin (5-HT) from human enterochromaffin cells and activates brain structures involved in nausea and vomiting

2011

Rotavirus (RV) is the major cause of severe gastroenteritis in young children. A virus-encoded enterotoxin, NSP4 is proposed to play a major role in causing RV diarrhoea but how RV can induce emesis, a hallmark of the illness, remains unresolved. In this study we have addressed the hypothesis that RV-induced secretion of serotonin (5-hydroxytryptamine, 5-HT) by enterochromaffin (EC) cells plays a key role in the emetic reflex during RV infection resulting in activation of vagal afferent nerves connected to nucleus of the solitary tract (NTS) and area postrema in the brain stem, structures associated with nausea and vomiting. Our experiments revealed that RV can infect and replicate in human…

RotavirusViral DiseasesViral Nonstructural ProteinsMiceChildlcsh:QH301-705.5Mice Inbred BALB CArea postremaBrainNauseaVagus NerveJejunumInfectious DiseasesMEDICINChild PreschoolEnterochromaffin cellVomitingMedicineSerotonin Antagonistsmedicine.symptomProto-Oncogene Proteins c-fosResearch Articlelcsh:Immunologic diseases. Allergymedicine.medical_specialtySerotoninVomitingImmunologyBiologyMicrobiologyRotavirus InfectionsSDG 3 - Good Health and Well-beingInternal medicineCell Line TumorVirologyGeneticsmedicineEnterochromaffin CellsAnimalsHumansBiologyMolecular BiologyGlycoproteinsToxins BiologicalMEDICINEVagus nerveEndocrinologyGene Expression Regulationlcsh:Biology (General)Cell cultureParasitologyEnteric nervous systemCalciumSerotoninlcsh:RC581-607Ex vivo
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Nicotinic and muscarinic modulation of 5-hydroxytryptamine (5-HT) release from porcine and canine small intestine

1992

Strips of porcine and canine small intestine were incubated in vitro and the release of 5-hydroxytryptamine (5-HT) was determined by HPLC with electrochemical detection. The spontaneous outflow of 5-HT from the porcine and canine small intestine largely reflects calcium-dependent 5-HT secretion from enterochromaffin cells which are under a spontaneous neuronal, excitatory input as indicated by the inhibitory effect (30-40%) of tetrodotoxin. In both species, nicotine enhanced the release of 5-HT in a concentration-dependent manner by a maximum of about 50% at 100 microM. This effect was blocked by the nicotine receptor antagonist hexamethonium, but not by the subtype-selective nicotine recep…

MaleNicotineSerotoninmedicine.medical_specialtySwineScopolamineHexamethonium CompoundsTetrodotoxinReceptors NicotinicBiologyHexamethoniumNicotine03 medical and health scienceschemistry.chemical_compoundDogs0302 clinical medicineInternal medicineIntestine SmallDrug DiscoveryMuscarinic acetylcholine receptorEnterochromaffin CellsmedicineOxotremorineAnimalsGenetics (clinical)030304 developmental biology0303 health sciencesMuscarineOxotremorineParasympatholyticsGeneral MedicineHydroxyindoleacetic AcidBungarotoxinsReceptors MuscarinicAcetylcholine3. Good healthNicotinic agonistEndocrinologyParasympathomimeticschemistryEnterochromaffin cellMolecular MedicineCalciumFemaleHexamethoniumDimethylphenylpiperazinium Iodide030217 neurology & neurosurgeryAcetylcholinemedicine.drugThe Clinical Investigator
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Cholinergic modulation of the release of 5-hydroxytryptamine from the guinea pig ileum.

1987

Isolated segments of the guinea pig ileum were vascularly perfused and the release of 5-HT and its metabolite 5-HIAA into the portal venous effluent determined by HPLC with electrochemical detection. Test substances were applied via the arterial perfusion medium. Oxotremorine inhibited concentration-dependently the release of 5-HT and 5-HIAA (by 47% at 1 mumol/l). Scopolamine (0.1 mumol/l) did not affect the release of 5-HT and 5-HIAA, but antagonized the effect of oxotremorine. In the presence of TTX (1 mumol/l), oxotremorine (1 mumol/l) increased the release of 5-HT by 150% and that of 5-HIAA by 220%. This increase was completely blocked by scopolamine. Hexamethonium (100 mumol/l) and TTX…

Malemedicine.medical_specialtySerotoninMetaboliteGuinea PigsScopolamineHexamethonium CompoundsBiologyIn Vitro TechniquesReceptors NicotinicHexamethoniumGuinea pigchemistry.chemical_compoundIleumInternal medicineMuscarinic acetylcholine receptorOxotremorinemedicineEnterochromaffin CellsAnimalsReceptors CholinergicIntestinal MucosaPharmacologyMuscarineOxotremorineGeneral MedicineEndocrinologynervous systemchemistryEnterochromaffin cellHexamethoniumSerotoninmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Modulation by 5-HT3 and 5-HT4 receptors of the release of 5-hydroxytryptamine from the guinea-pig small intestine.

1993

The effects of agonists and antagonists of 5-hydroxytryptamine (5-HT) receptors on the release of endogenous 5-HT from enterochromaffin cells were studied in the vascularly perfused isolated guinea-pig small intestine. The experiments were done in the presence of tetrodotoxin in order to exclude a neuronally mediated influence on 5-HT release. The 5-HT3 receptor agonist 2-methyl-5-HT increased 5-HT release, and this effect was antagonized by 1 nmol/l tropisetron. Nanomolar concentrations of tropisetron, MDL 72,222 and granisetron decreased 5-HT release. Ondansetron (0.1 and 1 mumol/l) did not modify 5-HT release. 5-Methoxytryptamine, BIMU8 and cisapride concentration-dependently inhibited 5…

AgonistMalemedicine.medical_specialtySerotoninmedicine.drug_classGuinea PigsStimulationTetrodotoxinBiologychemistry.chemical_compoundInternal medicineIntestine SmallmedicineEnterochromaffin CellsAnimalsIntestinal MucosaReceptorPharmacologyGeneral Medicinemusculoskeletal systemSerotonin Receptor AgonistsPerfusionEndocrinologychemistryMetitepineReceptors SerotoninAutoreceptorEnterochromaffin cellTropisetronFemaleSerotoninSerotonin Antagonistsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Evidence that neuronally released vasoactive intestinal polypeptide inhibits the release of serotonin from enterochromaffin cells of the guinea pig s…

1991

Abstract. Isolated small intestinal segments of the guinea pig were arterially perfused and the release of serotonin (5-hydroxytryptamine) and 5-hydroxyindoleacetic acid into the portal venous effluent was determined by HPLC with electrochemical detection. Test substances were intra-arterially applied. The muscarine receptor agonist oxotremorine (1 μmol/l inhibited the release of 5-hydroxytryptamine by about 50%. In the presence of the neurotoxin tetrodotoxin, oxotremorine enhanced the release of 5-hydroxytryptamine by 145%, indicating that the inhibitory effect of oxotremorine was mediated by the release of a neurotransmitter. Exogenous vasoactive intestinal polypeptide ( 1-100 pmol/l inhi…

MaleSerotoninmedicine.medical_specialtyEndocrinology Diabetes and MetabolismGuinea PigsVasoactive intestinal peptideTetrodotoxinBiologyAntibodiesGuinea pigchemistry.chemical_compoundEndocrinologyInternal medicineIntestine SmallEnterochromaffin CellsOxotremorinemedicineAnimalsNeurotransmitterChromatography High Pressure LiquidNeuronsMuscarineOxotremorineGeneral MedicineHydroxyindoleacetic AcidSmall intestineKineticsmedicine.anatomical_structureEndocrinologychemistryEnterochromaffin cellFemaleSerotoninVasoactive Intestinal Peptidemedicine.drugActa Endocrinologica
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